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Archive for the ‘Musculoskeletal’ Category

Frame Syndrome Osteomalacia and Receiving Social Security

Tuesday, January 17th, 2012

Osteoid is a bone protein matrix that is composed mainly of type 1 collagen. Osteoid builds up due to the fact that it does not mineralize properly when you have osteoblast or mineral dysfunction that is insufficient.

Your growth plate becomes irregular, thick and wide when the newly made bone of your growth plate does not mineralize. This results in a clinical diagnosis of rickets. This is true only in the case of children because adults do not have growth plates any longer.

Osteomalacia occurs when your remodeled bone does not mineralize. Osteomalacia is a condition that takes place in all ages. Many of the hereditary causes of osteomalacia show up during childhood and result in rickets.

Osteomalacia is characterized by a softening of your bones. In fact, that is what the meaning of osteomalacia is, “soft bones.”

Soft bones have a greater tendency to bow and fracture than harder, healthy bones do. When you have osteomalacia, your bone is prone to break down faster than it can re-form.

Osteomalacia and osteoporosis are not the same thing. Osteoporosis is another bone ailment that can also lead to bone fractures. Osteomalacia results from a defect in your bone-building process. Osteoporosis is caused by a weakening of previously constructed bone.

Frame syndrome is a rare kind of osteomalacia. It is a condition that is evidenced by an enzyme defect that affects bone formation. Frame syndrome mainly involves your ribs and your hip bone (iliac crest).

There is a racial predilection with frame syndrome. This condition takes place most of the time in Caucasians and blacks.

Frame syndrome is referred to in other ways. It is also called atypical osteomalacia involving the axial skeleton and axial osteomalacia.

In order for you to build strong, healthy bones, your body must have calcium and phosphate. Frame syndrome may develop if you do not receive enough of these two minerals from your diet or your body does not absorb these minerals properly.

There are several things that may result in these problems. Some of these things are:

  • Taking certain drugs that are used in the treatment of seizures, such as Phenobarbital and phenytoin
  • Having kidney or liver diseases
  • Having a Vitamin D deficiency
  • Having certain surgeries, such as gastrectomy (removing all or a part of your stomach)
  • Having an autoimmune disease that is known as celiac disease.

There are several signs and symptoms that you may have with frame syndrome. Some of these may include:

 

  • Tenderness that occurs over the area of your lumbar spine
  • A limited range of motion in the area of your spine
  • Back pain
  • Chronic axial pain that is vague
  • Fatigue that takes place in your extremities
  • Impairment in the formation of your bone
  • Osteomalacia of your hip bone (iliac crest)
  • Osteomalacia of your rib
  • Fragility of your bones
  • Weakness in your muscles
  • Weakness in your legs and arms
  • Diffuse body pains
  • A reduction in your ability to get around
  • A waddling gait when you walk.

 

 

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Inflammatory Fibrous Hyperplasia and Getting SSI or SSDI

Thursday, December 29th, 2011

Inflammatory fibrous hyperplasia is a chronic (continuing, ongoing) bone disease that is evidenced by a part of your bone developing abnormally. Scar-like (fibrous) tissue starts to replace and take the position of normal bone tissue. With the growth of your bone, the softer fibrous tissue keeps expanding, which leads to your bone becoming weakened by this process.

Deformity may occur in your bone that is infected by inflammatory fibrous hyperplasia. If deformity does develop, it will then increase the possibility of a fracture (break) occurring in your bone that has been affected. Do you have a severe case of inflammatory fibrous hyperplasia. If this is the case, you may be eligible to receive some form of social security disability benefits like SSI or SSDI. A smart move on your part would be to get in touch with one of the social security attorneys at socialsecurityhome.com to check this out. The social security attorneys at socialsecurityhome.com stand ready to assist you in obtaining all of the disability benefits that are rightfully yours.

Inflammatory fibrous hyperplasia is believed to begin before you are born. However, you may not realize that you are affected by inflammatory fibrous hyperplasia until you reach childhood, adolescence or adulthood.

About 7% of all benign bone tumors are due to this type of  hyperplasia. Your upper arm bone, skull, thighbone, shinbone and pelvis are the areas of your body where the disease occurs most often, but inflammatory fibrous hyperplasia may take place in any bone in your body.

In most cases, inflammatory fibrous hyperplasia involves only one of your bones. In these instances, it is known as monostotic inflammatory fibrous hyperplasia. When the disease affects two or more of your bones, it is called polystotic inflammatory fibrous hyperplasia. This form of the disease may affect two of your bones in the same limb or  several bones throughout your skeleton.

Men and women are affected equally by this. It also seems to affect all races equally.

Inflammatory fibrous hyperplasia is caused by a faulty (mutated) gene that has to do with your cells that produce bone. However, what causes this gene to become faulty is unknown.

What science does know is that inflammatory fibrous hyperplasia is neither inherited or passed down from parent to child. There are also no known environmental or dietary factors that lead to inflammatory fibrous hyperplasia.

Inflammatory fibrous hyperplasia is a disease that has no known cause. It develops spontaneously. This means that it does not result from another condition nor is it related to another disorder.

You might not have any signs and symptoms at all if your inflammatory fibrous hyperplasia is mild. If the disease is severe, however, you may experience several signs and symptoms. Some of these are:

  • Bone deformities
  • Problems with being able to walk
  • Bone sores (lesions)
  • Bone pain that grows worse with any type of activity but gets better when you rest
  • Pigmentation (an unusual skin color)
  • Bone fractures (breaks)
  • Problems with your endocrine gland
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Facet Arthrosis and Receiving Social Security Disability Benefits

Sunday, November 6th, 2011

Arthrosis is a term that comes from the Greek root word “arthros” that means “joint.” Arthrosis refers to a disease of your bone joint.

A facet joint is a small stabilizing joint that is located behind and between adjacent vertebrae. Nearly every movable or flexible level of your spine in your back is made up of vertebral body (bony building blocks of your spine), intervertebral discs that act like a cushion between each of your vertebral bodies and ties them together and facet joints.

Facet joints are found in the posterior section of your spine. Your spine is made up of 24 vertebrae. Two facet joints are located between the vertebrae of each spinal segment along your spinal column.

Facet arthrosis is a disorder that is marked by the degeneration or wear and tear that may take place on one or more of your facet joints. Facet arthrosis is a form of degenerative arthritis that involves the facet joints of your spine. The part of your spine where the facet joints are located is an area where arthritis pain may develop.

Facet arthrosis is known by other names. It is also referred to as facet joint syndrome, facet arthropathy and facet syndrome.

Facet arthrosis may develop in anyone at any time. However, facet arthrosis occurs most of the time in people who are elderly. Are you suffering from facet arthrosis? You may be able to get social security disability benefits, such as SSDI or SSI. You will never go wrong by contacting one of the social security attorneys at socialsecurityhome.com to see what your options are concerning disability benefits. The social security attorneys at socialsecurityhome.com can assist you with getting disability benefits.

At the present time, the exact cause of facet arthrosis has not been discovered. However, degeneration or wear and tear that takes place in your spine is usually the main thing that brings about this disorder. This is wear and tear or degeneration that comes about as a result of aging. There are other things that may also lead to facet arthrosis. Some of these are:

  • Torn ligaments that occur
  • Wear and tear that leads to a loss of cartilage between your joints that causes them to rub together
  • A previous back injury that you have had
  • Problems with your discs
  • Breaks (fractures) that you have sustained

There are some risk factors that may increase your risk of developing facet arthrosis. The primary risk factors are back injuries and aging.

Pain is the hallmark sign or symptom of facet arthrosis. The pain produced by facet arthrosis usually gets worse after you have been sleeping or resting. The pain may also be aggravated by twisting or bending backwards. This pain is usually located in your neck or lower back.

There are other signs and symptoms that you may experience with facet arthrosis. Some of these include:

  • Weakness, pain and numbness that occurs in your legs and buttocks
  • Headaches
  • Bone spurs
  • A reduced range of spinal movement
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Aseptic Necrosis and Receiving Social Security Disability

Saturday, October 1st, 2011
The head of the femur (Lat. caput femoris) wit...

Image via Wikipedia

Aseptic necrosis is a disease that comes from the temporary or permanent loss of the blood supply to a part of your bone that results in the death of bone tissue. This can then result in tiny breaks that take place in your bone and the eventual collapse of your bone. If this occurs near one of your joints, it may cause the collapse of the surface of your joint.

Aseptic necrosis can develop in anyone at any age. It may start in children or the elderly. However, it occurs most often in people who are in their 30s, 40s and 50s. Aseptic necrosis affects both men and women. However, it primarily affects men.

Somewhere around one in 27,000 people have aseptic necrosis in the United States. About 10,000 to 20,000 people acquire this disease each year in the United States.

Many of the cases of aseptic necrosis are the result of trauma to your bone, such as a dislocated joint or broken bone (fracture). This is because the trauma results in damage to your blood vessels that bring blood to your bone.

There are other causes of aseptic necrosis. Some of these include:

Ÿ  Decompression disease (the bends)

Ÿ  Lupus

Ÿ  Gaucher’s disease

Ÿ  Drinking alcohol to access

Ÿ  Sickle cell anemia

Ÿ  Radiation and chemotherapy treatments for cancer

Ÿ  Taking corticosteroids.

You may not exhibit any signs or symptoms in the early stages of aseptic necrosis, but the disease is progressive. What this means is that it gets worse with the passage of time.

The first sign or symptom that you may have with aseptic necrosis is joint pain. When the pain begins, you usually only experience it when you put weight on your affected joint. You may have pain even when you are at rest as aseptic necrosis progresses. You may also lose range of motion in your affected joint.

The pain usually begins gradually. It can range anywhere from mild to severe. Your pain may dramatically get worse if your bone and the surrounding surface of your joint collapse. Although the period of time from your first signs and symptoms to the loss of your joint function varies from person to person, it is usually anywhere from several months to over a year.

You or a loved one may be suffering from aseptic necrosis. Aseptic necrosis and/or complications that have arisen from it or other disorders that you have along with this disease may have led to the disability of you or your loved one and be the reason why you are not able to work.  Contact us today if you feel you need help to receive disability benefits from an experienced attorney.

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Axial Osteomalacia and Receiving Social Security Disability

Friday, September 30th, 2011
Osteoblasts actively synthesizing osteoid.

Image via Wikipedia

Osteoid is your bone protein matrix that is made up primarily of type 1 collagen. When there is insufficient mineral or osteoblast dysfunction, your osteoid accumulates because it does not mineralize properly.

When the newly formed bone of the growth plate does not mineralize, the growth plate becomes wide, irregular and thick. This leads to the clinical diagnosis of rickets. This is true only of children as adults no longer have growth plates. Osteomalacia develops when the remodeled bone does not mineralize. This occurs in all ages. Most of the hereditary causes of osteomalacia appear during childhood and cause rickets.

Osteomalacia is marked by a softening of your bones. In fact, the meaning of osteomalacia is “soft bones”.

Soft bones are more prone to bow and fracture than are harder, healthy bones. Your bone tends to break down faster than it can re-form in the case of osteomalacia.

Osteomalacia is not the same as osteoporosis. Osteoporosis is another bone disorder that can also result in bone fractures. Osteomalacia is caused by a defect in your bone-building process. Osteoporosis is due to a weakening of previously constructed bone.

Axial osteomalacia is a rare form of osteomalacia. It is a disorder that is marked by an enzyme defect that affects bone formation. Axial osteomalacia primarily affects your hip bone (iliac crest) and your ribs.

There is a racial predilection with axial osteomalacia. This disorder occurs predominantly in blacks and Caucasians.

Your body has to have phosphate and calcium in order to build healthy, strong bones. If your body does not absorb these minerals correctly or if you do not get enough of them in your diet, this may result in axial osteomalacia. Some of the things that may cause these difficulties include:

Ÿ  An autoimmune disease that is called celiac disease

Ÿ  Certain drugs that are used to treat seizures like Phenobarbital and phenytoin

Ÿ  Certain surgeries like gastrectomy (removing all or part of your stomach)

Ÿ  Liver or kidney diseases

Ÿ  Vitamin D deficiency.

There are several signs and symptoms that may be an indication of axial osteomalacia. Some of these are:

Ÿ  Impaired bone formation

Ÿ  Fatigue in your extremities

Ÿ  Limited range of motion in your spinal area

Ÿ  Vague chronic axial pain

Ÿ  Tenderness over your lumbar spine

Ÿ  Back pain.

You or a loved one may have axial osteomalacia. Axial osteomalacia and/or complications that have resulted from it or other conditions that you have in addition to this disorder may have caused the disability of you or your loved one and be what is preventing you from being able to work.

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Brittle Bone Disease and Receiving Social Security Disability

Tuesday, September 13th, 2011
X-ray of U.S. girl, age 16 years 8 months, wit...

Image via Wikipedia

Brittle bone disease is a skeletal disease that is evidenced by very fragile bones that fracture easily. These broken bones often take place under loads that normal bones stand up under without difficulty.

Brittle bone disease is a connective tissue disease. It results from a malfunction in your body’s production of the protein collagen. With brittle bone disease, the amount of the collagen that is made by your body is too little, or the quality is too poor.

Brittle bone disease is a fairly rare disease. Somewhere between 20,000 and 50,000 people in the United States are affected by this disease. Brittle bone disease develops with equal frequency among all ethnic and racial groups and among women and men.

There are four main types of brittle bone disease. Type I is the mildest and most common form of the disease. Signs and symptoms of Type I include:

Ÿ  Scoliosis (curvature of the spine)

Ÿ  Triangular-shaped face

Ÿ  Fragile bones

Ÿ  Hearing loss beginning in your 20s

Ÿ  Thin, smooth skin

Ÿ  Low muscle tone

Ÿ  Brittle teeth

Ÿ  Loose joints

Ÿ  Blue sclerae (whites of the eye).

Type II, brittle bone disease accounts for about 10% of the people with this disease.

This is the most severe form of the disease. This type of brittle bone disease often causes death at or shortly after birth.

About 20% of the people with brittle bone disease have Type III. People with this form of the disease frequently have 100 fractures by the time they reach puberty. Signs and symptoms of Type III are:

Ÿ  Sclera that have a purple, gray or blue tint

Ÿ  Curvature of the spine (scoliosis)

Ÿ  Loose joints

Ÿ  Possible respiratory problems

Ÿ  Soft bones that bend, as well as break, easily

Ÿ  Triangular-shaped face

Ÿ  Possible hearing loss

Ÿ  Barrel-shaped rib cage

Ÿ  Poor tooth development that often causes your teeth to be brittle and discolored

Ÿ  Short stature

Ÿ  Poor muscle development.

 

The severity of Type IV is somewhere between Type I and Type III. Breaks usually take place before puberty with the exception of women after menopause. Some of the signs and symptoms of brittle bone disease Type IV include:

Ÿ  Below average height

Ÿ  Mild to moderate bone deformity

Ÿ  Easily overstretched, loose joints

Ÿ  Scoliosis (curvature of the spine)

Ÿ  Barrel-shaped ribcage

Ÿ  Possible hearing loss

Ÿ  Triangular-shaped face

Ÿ  Possible brittle teeth.

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Degenerative Arthritis and Receiving Social Security Disability

Monday, September 12th, 2011
Arthrite rhumatoide Source: http://nihseniorhe...

Image via Wikipedia

Degenerative arthritis is a medical condition that is marked by low-grade inflammation that results in pain in your joints. Degenerative arthritis results from abnormal wearing of the cartilage that covers and acts like a cushion for your joints.

Degenerative arthritis is the most common kind of arthritis. Nearly 21 million people in the United States are afflicted with degenerative arthritis. About 25% of all the visits to the doctor’s office are because of this condition. Degenerative arthritis also accounts for around 50% of all non-steroidal, anti-inflammatory drug prescriptions.

There are two forms of degenerative arthritis. They are primary and secondary. Primary degenerative arthritis is a chronic degenerative condition that is related to aging, but it is not the result of aging. There are people well into their nineties who do not have any clinical or functional indications of degenerative arthritis. Secondary degenerative arthritis is due to other factors or diseases, but the medical results are the same as for primary degenerative arthritis.

Degenerative arthritis is also evidenced by the decrease or destruction of synovial fluid that lubricates your joints. You begin to experience pain upon weight bearing, including standing and walking, as your bone surfaces become not as well protected by cartilage. Your regional muscles may atrophy and your ligaments may become more lax because you have less movement due to the pain that you are experiencing.

Researchers believe that heredity may be a key factor in causing degenerative arthritis. This is because this condition often affects more than one member of the same family. Genetics play a role in the occurrence of degenerative arthritis, as well. There is also some evidence that allergies, whether fungal, infectious or systemically induced, may be a critical contributing factor in causing degenerative arthritis.

The hallmark sign or symptom of degenerative arthritis is chronic pain that leads to loss of mobility and possibly stiffness. The pain is usually evidenced by a burning sensation or a sharp ache in your surrounding tendons and muscles.

Degenerative arthritis can cause a crackling noise (called “crepitus”) as your affected joint is moved or touched, and you may have muscle spasm and contractions in your tendons.

At times, your joints may fill with fluid. Humid weather causes the pain to increase in many people. In theory, any joint in your body can be affected. However, degenerative arthritis usually affects your spine, hips, feet and hands.

You or a loved one may be afflicted with degenerative arthritis. Degenerative arthritis and/or complications that have been caused by it or other illnesses that you have besides this condition may have resulted in you or your loved one’s disability and inability to work.

If this is true, you may need assistance. You may need financial help.

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Barbeau’s Disease and Receiving Social Security Disability

Sunday, September 11th, 2011

Muscular dystrophy is a term that is used to designate a group of hereditary, genetic muscle diseases. These diseases lead to progressive muscle weakness in the muscles that enable you to move.

Muscular dystrophy means that you have missing or incorrect information in your genes. There are certain proteins that you need in order to have healthy muscles. Muscular dystrophy keeps these proteins from being produced. Muscular dystrophy is something that you inherit from your parents. It is not contagious. You cannot “catch it” from someone who has it.

Muscular dystrophy causes your muscles to get weak over time. People who are afflicted with muscular dystrophy may gradually lose the ability to do things that most of us take for granted like walking or sitting up. These problems that are caused by muscular dystrophy can begin at birth or shortly after. However, they can also take place later on in childhood, adolescence or adulthood.

There are several different forms of muscular dystrophy that affect different muscle groups in different ways. Barbeau’s disease is one of the kinds of muscular dystrophy.

Barbeau’s disease is usually an adult form of the disease. The onset of Barbeau’s disease is usually when you are in your 40s or 50s. It can even occur up to around the age of 70.

Little was known about what causes any kind of muscular dystrophy until the 1980s. Then, researchers found out that it is caused by a mutated (defective) gene. There is an insufficient amount of dystrophin because of the defective gene. Dystrophin is a protein that aids in keeping your muscle cells intact. In the case of Barbeau’s disease, there is a faulty gene that is believed to make extra chemical material that causes the development of clumps in your muscle cells.

Barbeau’s disease progresses slowly. The first sign or symptom of Barbeau’s disease is usually muscle weakness of your throat and eyelids. You may have difficulty swallowing. Your may have trouble keeping your eyes open or with drooping eyelids.

As Barbeau’s disease advances, you may have other signs and symptoms. Some of these include:

  • Characteristic face
  • Progressive muscle weakness in your limbs
  • Progressive weakness in your facial muscles
  • Progressive ptosis (drooping of your eyelids)
  • Weakness and wasting (loss of muscle tissue) of your tongue.

 

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Dystrophia Myotonica and Receiving Social Security Disability

Thursday, September 8th, 2011
Histopathology of gastrocnemius muscle from pa...

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Muscular dystrophy refers to a group of genetic (hereditary) muscle diseases. Progressive muscle weakness in your muscles that enable your body to move is what characterizes these diseases.

Muscular dystrophy involves incorrect or missing information in your genes. Proteins are stopped from being produced that are needed for healthy muscles. Muscular dystrophy is a disease that is passed down to you from your parents. It is not something that you can catch from someone who has it. Muscular dystrophy is not contagious.

Muscular dystrophy weakens your muscles with the passage of time. You may gradually lose your ability to do things that most people do not even think about, like sitting up or walking. These difficulties may have originated when you were a baby, or they can start in childhood, adolescence or adulthood.

There are several different kinds of muscular dystrophy that affect different muscle groups in different ways. Dystrophia myotonica is one of the forms of this disease.

Dystrophia myotonica is the most common kind of muscular dystrophy that starts in adulthood. It usually develops between the ages of 20 and 40. However, there is an infantile form of dystrophia myotonica. In fact, this disease can begin at any age from birth to old age.

Until the 1980s, there was not much known about what causes any type of muscular dystrophy. Then, researchers discovered that muscular dystrophy is caused by a defective gene. Not enough dystrophin is made because of the faulty gene. Dystrophin is a protein that helps keep your muscle cells intact. In the case of dystrophia myotonica, there is a repeated section of DNA on either chromosome 3 or chromosome 19.

The progression of dystrophia myotonica is slow. The disease can span 50 to 60 years.

The first signs and symptoms of dystrophia myotonica are usually generalized weakness and loss of muscle tissue (muscle wasting) in your hands, forearms, lower legs, face and neck. This is in conjunction with difficulty relaxing muscles after contracting them. Other signs and symptoms that you may experience are:

  • Mild diabetes
  • Difficulty breathing and swallowing
  • Frontal balding in men
  • Dizziness or fainting
  • Inability to concentrate
  • Problems with your digestive tract like diarrhea and constipation
  • Daytime sleepiness
  • Difficulty sleeping well at night
  • Clouding of the lenses of your eyes (cataracts).

You or your loved one may have decided to apply for the financial help that you need from the Social Security Administration by applying for Social Security disability benefits or disability benefits because of the disability that has resulted from dystrophia myotonica and/or complications that have been brought about by it or other ailments that you have in conjunction with this disease. You may have already applied and been denied by the Social Security Administration.

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MD and Receiving Social Security Disability Benefits

Thursday, September 8th, 2011
Collage of varius Gray's muscle pictures by Mi...

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Most people have heard of MD (muscular dystrophy) because of the Labor Day telethon that is held each year. However, other than this, most people do not know much about MD.

 

MD refers to more than one single disease. MD is used for a group of hereditary (genetic) muscle diseases that result in progressive muscle weakness in the muscles that enable your body to move.

People with MD have incorrect or missing information in their genes that keep them from producing the proteins that are required for healthy muscles. Because MD is congenital (something you are born with), it is not something that is contagious, that you can get from someone who has it.

MD weakens your muscles over time. Children, teens and adults with this disease may gradually lose their ability to do the things that most people take for granted, like walking or sitting up. The muscle problems resulting from MD can begin when you were a baby, or they may start later on. Some adults acquire MD.

There are several different kinds of MD that can affect different muscle groups in different ways. Some of these include Duchenne MD, distal MD, Becker’s MD, myotonic MD, congenital MD, limb-girdle MD, facioscapulohumeral MD, oculopharyngeal MD and Emery-Dreifuss MD.

Somewhere around 250,000 children and adults have MD in the United States. Some forms of MD like Becker’s and Duchenne affect primarily boys.

As mentioned earlier, MD is a genetic (inherited) disease. This means that it is passed down through families. It is caused by defects (mutations) that occur in the genes that are passed down to you from your parents. Each one of the various kinds of MD is caused by a genetic mutation that is particular to that form of the disease.

There are many signs and symptoms that you may experience with MD. They will depend on the type of MD that you have. Some possible signs and symptoms include:

  • Frequent falls
  • Progressive muscular weakness or wasting
  • Poor balance
  • Pain in your calf
  • Drooping eyelids
  • Walking difficulty or waddling gait
  • The inability to walk
  • Gonadal atrophy
  • Muscle contractures
  • Scoliosis
  • Respiratory problems
  • Limited range of movement.

Some types of MD can also affect your heart. These kinds of MD can cause arrhythmias or cardiomyopathy.

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